tallózása szerző szerint "Castelo-Branco, P"

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  • Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma 
    Schwartzentruber, J; Korshunov, A; Liu, XY; Jones, DT; Pfaff, E; Jacob, K; Sturm, D; Fontebasso, AM; Quang, DA; Tonjes, M; Hovestadt, V; Albrecht, S; Kool, M; Nantel, A; Konermann, C; Lindroth, A; Jager, N; Rausch, T; Ryzhova, M; Korbel, JO; Hielscher, T; Hauser, Péter; Garami, Miklós; Klekner, Álmos; Bognár, László; Ebinger, M; Schuhmann, MU; Scheurlen, W; Pekrun, A;; Fruhwald, MC; Roggendorf, W; Kramm, C; Durken, M; Atkinson, J; Lepage, P; Montpetit, A; Zakrzewska, M; Zakrzewski, K; Liberski, PP; Dong, Z; Siegel, P; Kulozik, AE; Zapatka, M; Guha, A; Malkin, D; Felsberg, J; Reifenberger, G; von Deimling, A; Ichimura, K; Collins, VP; Witt, H; Milde, T; Witt, O; Zhang, C; Castelo-Branco, P; Lichter, P; Faury, D; Tabori, U; Plass, C; Majewski, J; Pfister, SM; Jabado, N (2012)
    Glioblastoma multiforme (GBM) is a lethal brain tumour in adults and children. However, DNA copy number and gene expression signatures indicate differences between adult and paediatric cases. To explore the genetic events ...
  • Subgroup-Specific Prognostic Implications of TP53 Mutation in Medulloblastoma 
    Zhukova N; Ramaswamy V; Remke M; Pfaff E; Shih DJ; Martin, DC; Castelo-Branco, P; Baskin, B; Ray, PN; Bouffet, E; von Bueren, AO; Jones, DT; Northcott, PA; Kool, M; Sturm, D; Pugh, TJ; Pomeroy, SL; Cho, YJ; Pietsch, T; Gessi, M; Rutkowski, S; Bognár, László; Klekner, Álmos; Cho, BK; Kim, SK; Wang, KC; Eberhart, CG; Fevre-Montange, M; Fouladi, M; French, PJ; Kros, M; Grajkowska, WA; Gupta, N; Weiss, WA; Hauser, Péter; Jabado, N; Jouvet, A; Jung, S; Kumabe, T; Lach, B; Leonard, JR; Rubin, JB; Liau, LM; Massimi, L; Pollack, IF; Shin, Ra Y; Van, Meir EG; Zitterbart, K; Schuller, U; Hill, RM; Lindsey, JC; Schwalbe, EC; Bailey, S; Ellison, DW; Hawkins, C; Malkin, D; Clifford, SC; Korshunov, A; Pfister, S; Taylor, MD; Tabori, U (2013)
    PURPOSE Reports detailing the prognostic impact of TP53 mutations in medulloblastoma offer conflicting conclusions. We resolve this issue through the inclusion of molecular subgroup profiles. PATIENTS AND METHODS We ...