tallózása szerző szerint "Dóczi, Judit"

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Abolition of mitochondrial substrate-level phosphorylation by itaconic acid produced by LPS-induced Irg1 expression in cells of murine macrophage lineage

Németh, Beáta; Dóczi, Judit; Csete, Dániel; Kacsó, Gergely; Ravasz, Dóra; Daniel Adams; Kiss, Gergely; Nagy, Ádám Miklós; Horváth, Gergő; Tretter, László; Mócsai, Attila; Csépányi-Kömi, Roland; Iordanov, Iordan; Ádám, Veronika; Chinopoulos, Christos (2016)

Itaconate is a nonamino organic acid exhibiting antimicrobial effects. It has been recently identified in cells of macrophage lineage as a product of an enzyme encoded by immunoresponsive gene 1 (Irg1), acting on the citric ...
Alterations in voltage-sensing of the mitochondrial permeability transition pore in ANT1-deficient cells

Dóczi, Judit; Törőcsik, Beáta; Echaniz-Laguna A; Mousson de Camaret B; Starkov A; Starkova N; Gál, Anikó; Molnár, Mária Judit; Kawamata H; Manfredi G; Ádám, Veronika; Chinopoulos, Christos (2016)

The probability of mitochondrial permeability transition (mPT) pore opening is inversely related to the magnitude of the proton electrochemical gradient. The module conferring sensitivity of the pore to this gradient has ...
BGP-15 inhibits caspase-independent programmed cell death in acetaminophen-induced liver injury

Nagy G,; Szarka, András; Lotz, Gábor; Dóczi, Judit; Wunderlich, Lívius; Kiss, András; Jemnitz, Katalin; Veres, Zsuzsa; Bánhegyi, Gábor; Schaff, Zsuzsa; Sümegi, Balázs; Mandl, József (2010)

It has been recently shown that acute acetaminophen toxicity results in endoplasmic reticulum redox stress and an increase in cells with apoptotic phenotype in liver. Since activation of effector caspases was absent, the ...
Exclusive neuronal detection of KGDHC-specific subunits in the adult human brain cortex despite pancellular protein lysine succinylation

Dobolyi, Árpád; Bagó, Attila György; Palkovits, Miklós; Nemeria, NS; Jordan, F; Dóczi, Judit; Ambrus, Attila; Ádám, Veronika; Chinopoulos, Christos (2020)
Mutated SUCLG1 causes mislocalization of SUCLG2 protein, morphological alterations of mitochondria and an early-onset severe neurometabolic disorder

Chinopoulos, Christos; Batzios S; van den Heuvel L.P; Rodenburg R; Smeets R; Waterham HR; Turkenburg M; Ruiter JP; Wanders RJA; Dóczi, Judit; Horvath G; Dobolyi, Árpád; Vargiami E; Wevers RA; Zafeiriou D (2018)
The Mitochondrial Targets of Neuroprotective Drug Vinpocetine on Primary Neuron Cultures, Brain Capillary Endothelial Cells, Synaptosomes, and Brain Mitochondria

Sváb, Gergely; Dóczi, Judit; Gerencser, Akos A; Ambrus, Attila; Gallyas, Ferenc; Sümegi, Balázs; Tretter, László (2019)

Vinpocetine is considered as neuroprotectant drug and used for treatment of brain ischemia and cognitive deficiencies for decades. A number of enzymes, channels and receptors can bind vinpocetine, however the mechanisms ...
Two transgenic mouse models for beta subunit components of succinate-CoA ligase yielding pleiotropic metabolic alterations

Kacsó, Gergely; Ravasz, Dóra; Dóczi, Judit; Németh, Beáta; Madgar O,; Iordanov, Iordan; Gál, Anikó; Molnár, Mária Judit; Nagy, Zsolt; Patócs, Attila Balázs; Ádám, Veronika; Chinopoulos, Christos (2016)

Succinate-CoA ligase is a heterodimer enzyme composed of Suclg1 -alpha- and a substrate-specific Sucla2 or Suclg2 -beta- subunit yielding ATP or GTP, respectively. In humans, the deficiency of this enzyme leads to ...

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tallózása szerző szerint "Dóczi, Judit"

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