tallózása szerző szerint "MTA-SE Neurobiokémiai és Molekuláris Élettani Kutatócsoport (2006-ig: MTA-SE Neurobiokémiai Kutatócsoport) [2006.12.31]"

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Paracrine transactivation of the CB1 cannabinoid receptor by AT1 angiotensin and other Gq/11 protein-coupled receptors.

Turu Gábor; Várnai Péter; Gyombolai Pál; Szidonya László; Offertaler L; Bagdy György; Hunyady László (2009)

Intracellular signaling systems of G protein-coupled receptors are well established, but their role in paracrine regulation of adjacent cells is generally considered as a tissue-specific mechanism. We have shown ...
Personalized medicine can pave the way for the safe use of CB(1) receptor antagonists.

Lazáry Judit; Juhász Gabriella; Hunyady László; Bagdy György (2011)

Antagonists of cannabinoid type-1 (CB(1)) receptors have been explored as therapeutic agents for obesity and addiction. However, use of rimonabant (the first marketed CB(1) receptor antagonist) has been suspended due to ...
Regulation of endocannabinoid release by G proteins: A paracrine mechanism of G protein-coupled receptor action.

Gyombolai Pál; Pap Dorottya; Turu Gábor; Catt KJ; Bagdy György; Hunyady László (2012)

In the past years, the relationship between the endocannabinoid system (ECS) and other hormonal and neuromodulatory systems has been intensively studied. G protein-coupled receptors (GPCRs) can stimulate endocannabinoid ...
The role of diacylglycerol lipase in constitutive and angiotensin AT(1) receptor-stimulated cannabinoid CB1 receptor activity

Turu, Gáor; Simon A; Gyombolai Pál; Szidonya László; Bagdy György; Hunyady László (2007)

The cannabinoid CB1 receptor (CB1R) is,a G protein-coupled receptor, which couples to the G(i/o) family of heterotrimeric G proteins. The receptor displays both basal and agonist-induced signaling and internalization. ...

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tallózása szerző szerint "MTA-SE Neurobiokémiai és Molekuláris Élettani Kutatócsoport (2006-ig: MTA-SE Neurobiokémiai Kutatócsoport) [2006.12.31]"

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tallózása szerző szerint "MTA-SE Neurobiokémiai és Molekuláris Élettani Kutatócsoport (2006-ig: MTA-SE Neurobiokémiai Kutatócsoport) [2006.12.31]"

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