tallózása szerző szerint "Li S"

1-7 / 7

Administration of zinc complex of acetylsalicylic acid after the onset of myocardial injury protects the heart by upregulation of antioxidant enzymes

Korkmaz-Icoz S; Atmanli A; Radovits, Tamás; Li S; Hegedus P; Ruppert, Mihály; Brlecic P; Yoshikawa Y; Yasui H; Karck M; Szabó, Gábor Balázs (2016)

We recently demonstrated that the pre-treatment of rats with zinc and acetylsalicylic acid complex in the form of bis(aspirinato)zinc(II) [Zn(ASA)2] is superior to acetylsalicylic acid in protecting the heart from acute ...
An altered pattern of myocardial histopathological and molecular changes underlies the different characteristics of type-1 and type-2 diabetic cardiac dysfunction.

Radovits, Tamás; Korkmaz S; Mátyás, Csaba; Oláh, Attila; Németh, Balázs Tamás; Pali S; Hirschberg K; Zubarevich A; Gwanmesia PN; Li S; Loganathan S; Barnucz, Enikö; Merkely, Béla Péter; Szabó, Gábor Balázs (2015)

Increasing evidence suggests that both types of diabetes mellitus (DM) lead to cardiac structural and functional changes. In this study we investigated and compared functional characteristics and underlying subcellular ...
Dimethyloxalylglycine treatment of brain-dead donor rats improves both donor and graft left ventricular function after heart transplantation.

Hegedűs, Péter; Li S; Korkmaz-Icöz S; Radovits, Tamás; Mayer T; Al Said S; Brlecic P; Karck M; Merkely, Béla Péter; Szabó, Gábor Balázs (2016)

Abstract OBJECTIVE: Hypoxia inducible factor (HIF)-1 pathway signalling has a protective effect against ischemia/reperfusion injury. The prolyl-hydroxylase inhibitor dimethyloxalylglycine (DMOG) activates the HIF-1 pathway ...
Mild Type 2 Diabetes Mellitus Reduces the Susceptibility of the Heart to Ischemia/Reperfusion Injury: Identification of Underlying Gene Expression Changes.

Korkmaz-Icoz S; Lehner A; Li S; Vater A; Radovits, Tamás; Hegedűs, Péter; Ruppert, Mihály; Brlecic P; Zorn M; Karck M; Szabó, Gábor Balázs (2015)

Despite clinical studies indicating that diabetic hearts are more sensitive to ischemia/reperfusion injury, experimental data is contradictory. Although mild diabetes prior to ischemia/reperfusion may induce a myocardial ...
Oral treatment with a zinc complex of acetylsalicylic acid prevents diabetic cardiomyopathy in a rat model of type-2 diabetes: activation of the Akt pathway.

Korkmaz-Icoz S; Al Said S; Radovits, Tamás; Li S; Brune M; Hegedus P; Atmanli A; Ruppert, Mihály; Brlecic P; Lehmann LH; Lahrmann B; Grabe N; Yoshikawa Y; Yasui H; Most P; Karck M; Szabó, Gábor Balázs (2016)

BACKGROUND: Type-2 diabetics have an increased risk of cardiomyopathy, and heart failure is a major cause of death among these patients. Growing evidence indicates that proinflammatory cytokines may induce the development ...
Q50, an Iron-Chelating and Zinc-Complexing Agent, Improves Cardiac Function in Rat Models of Ischemia/Reperfusion-Induced Myocardial Injury.

Korkmaz S; Barnucz E; Loganathan S; Li S; Radovits Tamás; Puskás László; Faragó Nóra; Gyuris Márió; Merkely Béla Péter (2013)

Background: Reperfusion of ischemic myocardium may contribute to substantial cardiac tissue damage, but the addition of iron chelators, zinc or zinc complexes has been shown to prevent heart from reperfusion injury. We ...
Total Aortic Arch Replacement: Superior Ventriculo-Arterial Coupling with Decellularized Allografts Compared with Conventional Prostheses.

Weymann A; Radovits, Tamás; Schmack B; Korkmaz S; Li S; Chaimow N; Patzold I; Becher PM; Hartyánszky, István; Soós, Pál; Merkely, Gergő; Németh, Balázs Tamás; Istók, Roland; Veres, Gábor; Merkely, Béla Péter; Terytze K; Karck M; Szabo G (2014)

BACKGROUND: To date, no experimental or clinical study provides detailed analysis of vascular impedance changes after total aortic arch replacement. This study investigated ventriculoarterial coupling and vascular impedance ...

1-7 / 7

Keresés

Összetett keresés

Tallózás

Teljes repozitórumban

Hozzáférésem

Bejelentkezés

tallózása szerző szerint "Li S"

DSpace/Manakin Repository

tallózása szerző szerint "Li S"

Keresés

Tallózás

Teljes repozitórumban

Hozzáférésem