tallózása szerző szerint "Maros ME"

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Cell cycle analysis can differentiate thin melanomas from dysplastic nevi and reveals accelerated replication in thick melanomas.

Kiszner, Gergő; Wichmann, Barnabás; Németh, István Balázs; Varga, Erika; Meggyesházi, Nóra; Teleki, Ivett; Balla, Péter; Maros ME; Penksza, Károly; Krenács, Tibor (2014)

Cell replication integrates aberrations of cell cycle regulation and diverse upstream pathways which all can contribute to melanoma development and progression. In this study, cell cycle regulatory proteins were detected ...
Prognostic impact of reduced connexin43 expression and gap junction coupling of neoplastic stromal cells in giant cell tumor of bone

Balla, Péter; Maros ME; Barna, Gábor; Antal, Imre; Papp, Gergő; Sápi, Zoltán; Athanasou NA; Benassi MS; Picci P; Krenács, Tibor (2015)

Missense mutations of the GJA1 gene encoding the gap junction channel protein connexin43 (Cx43) cause bone malformations resulting in oculodentodigital dysplasia (ODDD), while GJA1 null and ODDD mutant mice develop osteopenia. ...
Upregulation of heat shock proteins and the promotion of damage-associated molecular pattern signals in a colorectal cancer model by modulated electrohyperthermia

Andocs G; Meggyesházi, Nóra; Balogh, Lajos; Spisák, Sándor; Maros ME; Balla, Péter; Kiszner, Gergő; Teleki, Ivett; Kővágó, Csaba; Krenács, Tibor (2015)

In modulated electrohyperthermia (mEHT) the enrichment of electric field and the concomitant heat can selectively induce cell death in malignant tumors as a result of elevated glycolysis, lactate production (Warburg ...

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tallózása szerző szerint "Maros ME"

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