tallózása szerző szerint "Shah YM"

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Iron Uptake via DMT1 Integrates Cell Cycle with JAK-STAT3 Signaling to Promote Colorectal Tumorigenesis

Xue X; Ramakrishnan SK; Weisz K; Triner D; Xie L; Pant A; Attili D; Győrffy, Balázs; Zhan M; Carter-Su C; Hardiman KM; Wang TD; Dame MK; Varani J; Brenner D; Fearon ER; Shah YM (2016)

Dietary iron intake and systemic iron balance are implicated in colorectal cancer (CRC) development, but the means by which iron contributes to CRC are unclear. Gene expression and functional studies demonstrated that the ...
Transcription factor ZBP-89 drives a feedforward loop of beta-catenin expression in colorectal cancer

Essien BE; Sundaresan S; Ocadiz-Ruiz R; Chavis A; Tsao AC; Tessier AJ; Hayes MM; Photenhauer A; Saqui-Salces M; Kang AJ; Shah YM; Győrffy, Balázs; Merchant JL (2016)

In colorectal cancer (CRC), APC-mediated induction of unregulated cell growth involves post-translational mechanisms that prevent proteasomal degradation of proto-oncogene beta-catenin (CTNNB1) and its eventual translocation ...
Tumor-selective proteotoxicity of verteporfin inhibits colon cancer progression independently of YAP1

Zhang H; Ramakrishnan SK; Triner D; Centofanti B; Maitra D; Győrffy, Balázs; Sebolt-Leopold JS; Dame MK; Varani J; Brenner DE; Fearon ER; Omary MB; Shah YM (2015)

Yes-associated protein 1 (YAP1) is a transcriptional coactivator in the Hippo signaling pathway. Increased YAP1 activity promotes the growth of tumors, including that of colorectal cancer (CRC). Verteporfin, a drug that ...

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tallózása szerző szerint "Shah YM"

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