Vasculopathiak autoimmun vonatkozásai

dc.contributor.author	Soltész, Pál
dc.contributor.author	Prohászka, Zoltán
dc.contributor.author	Füst, György
dc.contributor.author	Der H
dc.contributor.author	Kerekes, György
dc.contributor.author	Szodoray P
dc.contributor.author	Zeher, Margit
dc.contributor.author	Szekanecz, Zoltán
dc.date.accessioned	2018-10-10T09:43:45Z
dc.date.available	2018-10-10T09:43:45Z
dc.date.issued	2007
dc.identifier	34547654447
dc.identifier.citation	pagination=53-57; journalVolume=148; journalIssueNumber=Suppl. 1; journalTitle=ORVOSI HETILAP;
dc.identifier.uri	http://repo.lib.semmelweis.hu//handle/123456789/6396
dc.identifier.uri	doi:10.1556/OH.2007.28036
dc.description.abstract	In the development of atherosclerotic plaques we can identify three autoantigens, the pathological value of which has been proven by experimental and clinical data. These antigens are the 60 kDa heat-shock-protein, ?2-glycoprotein I and oxidized LDL. They have role in the antigen-specific T-cell differentiation processes, moreover, against these antigens autoantibodies are produced, which have prothrombotic activity, leading to the acceleration of atherosclerosis. In autoimmune diseases besides these factors other mechanisms are present, which lead to the development of autoimmune vasculopathies. The current review gives an overview of these vasculopathies.
dc.relation.ispartof	urn:issn:0030-6002
dc.title	Vasculopathiak autoimmun vonatkozásai
dc.type	Journal Article
dc.date.updated	2018-09-02T10:33:45Z
dc.language.rfc3066	hu
dc.identifier.mtmt	1132240
dc.identifier.pubmed	17430795
dc.contributor.department	DE/KK/BelgyKL/C épület
dc.contributor.institution	Debreceni Egyetem