tallózása szerző szerint "Liberski PP"

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  • Erratum: Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma (Nature (2012) 482 (226-231)) 
    Schwartzentruber J; Korshunov A; Liu X-Y; Jones DTW; Pfaff E; Jacob K; Sturm D; Fontebasso AM; Quang D-AK; Tönjes M; Hovestadt V; Albrecht S; Kool M; Nantel A; Konermann C; Lindroth A; Jäger N; Rausch T; Ryzhova M; Korbel JO; Hielscher T; Hauser, Péter; Garami, Miklós; Klekner, Álmos; Bognár, László; Ebinger M; Schuhmann MU; Scheurlen W; Pekrun A; Frühwald MC; Roggendorf W; Kramm C; Dürken M; Atkinson J; Lepage P; Montpetit A; Zakrzewska M; Zakrzewski K; Liberski PP; Dong Z; Siegel P; Kulozik AE; Zapatka M; Guha A; Malkin D; Felsberg J; Reifenberger G; Von Deimling A; Ichimura K; Collins VP; Witt H; Milde T; Witt O; Zhang C; Castelo-Branco P; Lichter P; Faury D; Tabori U; Plass C; Majewski J; Pfister SM; Jabado N (2012)
  • Molecular Profiling Identifies Prognostic Subgroups of Pediatric Glioblastoma and Shows Increased YB-1 Expression in Tumors. 
    Faury D; Nantel A; Dunn SE; Guiot MC; Haque T; Hauser, Péter; Garami, Miklós; Bognár, László; Hanzély Z; Liberski PP; Lopez-Aguilar E; Valera ET; Tone LG; Carret AS; Del Maestro RF; Gleave M; Montes JL; Pietsch T; Albrecht S; Jabado N (2007)
    PURPOSE: Pediatric glioblastoma (pGBM) is a rare, but devastating brain tumor. In contrast to GBM in adults (aGBM), little is known about the mechanisms underlying its development. Our aim is to gain insight into the ...
  • Mutations in SETD2 and genes affecting histone H3K36 methylation target hemispheric high-grade gliomas 
    Fontebasso AM; Schwartzentruber J; Khuong-Quang D-A; Liu X-Y; Sturm D; Korshunov A; Jones DTW; Witt H; Kool M; Albrecht S; Fleming A; Hadjadj D; Busche S; Lepage P; Montpetit A; Staffa A; Gerges N; Zakrzewska M; Zakrzewski K; Liberski PP; Hauser, Péter; Garami, Miklós; Klekner, Álmos; Bognár, László; Zadeh G; Faury D; Pfister SM; Jabado N; Majewski J (2013)
    Recurrent mutations affecting the histone H3.3 residues Lys27 or indirectly Lys36 are frequent drivers of pediatric high-grade gliomas (over 30 % of HGGs). To identify additional driver mutations in HGGs, we investigated ...