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dc.contributor.author Stark, Júlia
dc.contributor.author Tulassay, Zsolt
dc.contributor.author Lengyel, Gabriella
dc.contributor.author Szombath, Dezső
dc.contributor.author Székács, Béla
dc.contributor.author Adler, Ildikó
dc.contributor.author Marczell, István
dc.contributor.author Nagy-Repas P
dc.contributor.author Dinya, Elek
dc.contributor.author Rácz, Károly
dc.contributor.author Békési, Gábor
dc.date.accessioned 2018-05-23T12:43:32Z
dc.date.available 2018-05-23T12:43:32Z
dc.date.issued 2013
dc.identifier 84879208152
dc.identifier.citation pagination=84-88; journalVolume=100; journalIssueNumber=1; journalTitle=ACTA PHYSIOLOGICA HUNGARICA;
dc.identifier.uri http://repo.lib.semmelweis.hu//handle/123456789/5180
dc.identifier.uri doi:10.1556/APhysiol.99.2012.001
dc.description.abstract Background: In our earlier studies both corticosterone and cortisol had antioxidant effect in vitro.Objectives: Our aim was to clarify whether corticosterone and cortisol oral administration results in beneficial antioxidant changes in Sprague-Dawley adult male rats in vivo.Methods: Experimental animals were fed a lipid rich diet and treated with corticosterone or cortisol in the drinking fluid. Control group was fed only lipid rich diet with untreated drinking water. The untreated group was fed a normal diet with untreated water. Total scavenger capacity (TSC) was measured before and after 4 weeks of treatment in blood samples using a chemiluminometric assay.Results: Both corticosterone and cortisol treatment caused increased TSC. The control group and the untreated group showed no significant changes in TSC. Conclusion: Our results support the hypothesis that corticosterone and cortisol administration can improve the antioxidant status not only in vitro but also in vivo.
dc.relation.ispartof urn:issn:0231-424X
dc.title Increased total scavenger capacity in rats fed corticosterone and cortisol on lipid-rich diet.
dc.type Journal Article
dc.date.updated 2018-03-17T21:40:53Z
dc.language.rfc3066 en
dc.identifier.mtmt 2163894
dc.identifier.wos 000316679000008
dc.identifier.pubmed 23232702
dc.contributor.department SE/AOK/I/Kórélettani Intézet
dc.contributor.department SE/AOK/K/II. Sz. Belgyógyászati Klinika
dc.contributor.department SE/EKK/Egészségügyi Informatikai Fejlesztő és Továbbképző Intézet [2015.06.30]
dc.contributor.institution Semmelweis Egyetem
dc.mtmt.swordnote A Teljes dokumentumon és a PubMedben nem szerepel utolsó szerzőként Horváth Iván.


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